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L-Carnitine works as a shuttle in both directions removing the excess of Acyls units from inside the mitochondria. Any accumulation of those products and excessive Acyls units can disturb and perturbate the mitochondrial function. L-Carnitine is a powerful antioxidant, detoxify ammonia, neutralize lactic acidosis and is an iron-chelator.

Co-Q can neutralize all ROS created inside the mitochondria and elsewhere in the cell and outside cell, and as a matter of fact, is a potent anti-oxydant who protects against mitochondrial decay.

Co-Q makes lipid membranes more resistant to peroxydation, reduces blood pressure and decrease CRP. Another important role for Ubiquinone is to stabilize membranes and tyren vitler anti aging, it works as anti-arrhytmic agent.

While blocking cholesterol synthesis, it can inhibit Co-Q production at the same time mevalonic ac pathway. Involved in many as enzyme-reactions in the body. Is essential for the transport of ATP in the cytosol, helps to relax the muscle walls of the arteries, acts as an calcium channel blocker, as a result, decreases blood pressure.

Magnesium is used for arryhtmias and mitral valve prolapsus. IV administration is used in a variety of cardiac emergencies, improve NOs activity, and as result, increase coronary blood flow. Works very well against migraine orally, mg bid daily and IV in case of acute crisis. Those lipoproteins after a lipid-rich dinner f. Those fat particles are among the most potent causes of heart attacks and strokes.

Normally, in healthy adults, those particles should be cleared from the blood within 4 to 6 hours. Many clinical studies have indicated that those postprandial lipoproteins are powerful instigators of coronary plaque, carotid plaque and plaque in the aorta.

In reality, they block NO production, increase releasing of Endothelin by EC, causing Endothelial dysfunction, increase cell adhesion molecules CAM allowing white blood cells to adhere and enter genius ultimate peeling anti-îmbătrânire în două faze arterial wall, leading to plaque formation, and activate blood clotting factors while inhibiting clot breakdown. Last, they trigger the production of small LDL particles.

D-Ribose is a 5 carbons sugar pentoseand is not a part of glycolysis 6C.

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Synthethized from glucose through an independent mechanism called the Pentose Phosphate Pathway which is: - Time consuming - Rate-limited by 2 enzymes: GP-D glucosephosphate dehydrogenase 6-P-G-D 6phosphogluconate dehydrogenase Supplementing with D-Ribose bypasses this slow and rate limited pathway, will inevitably accelerate ATP synthesis.

This pentose is quickly absorbed orally, 95 per cent of it will be used by different tissues while only 5 per cent will be excreted in the urine. No IDL Intermediate density lipoprotein should be present in blood draw fasting.

How to lower PPHD? Tocotrienols can decrease LDL at a dosage of 75 mg a day. During this study, took low to moderate doses of Aspirin.

The conclusion was very clear: - Women who reported taking low to moderate doses of aspirin had a 25 per cent lower risk of death from any cause.

It speaks for itself. Inflammation causes arterial plaques to breakoff and plays a deadly role in destabilizing arterial plaques. Meanwhile, it can cause excessive free radical damage, therefore it can deplete the body's reserves in antioxidants.

New strategy to cool inflammation -protocol- : - Use of Boswellia extract to inhibit 5-lox 5-loxin.

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Vit D: Down regulate gene expression of NF-Kappa Bta key role in inflammation and cancer and has a powerful immunomodulator activity. Anti-Inflammatory diet: Rich in nuts, cereals, MU fats, fish, Colza oil, low glycemic index, low in arachidonic acid and rich in fibers.

Homocystein, the silent killer: Homocysteine is a byproduct of the metabolism of Methionine. High plasma level of homocysteine is a powerful risk factor for MI,stroke, Alzheimer, depression osteoporosis, etcand is an independent risk factor for CVD. You can reduce your homocysteine by enhancing methylation, by transferring a methyl group-CH3 from one molecule to another. Methylation takes place in every cell several times daily. When Methionine donates her methyl group, it becomes Homocysteine.

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They can facilitate the recycling of homocysteine back to Methionine. This is what we call a Methylation process. This reaction does need the help of methylating factors as, Vit B6, Vit B12, folic ac and zinc. Homocystein is causing the initial damage to the lining of the blood vessels, opening the door to oxidized LDL to penetrate the intima and build up inside the walls with macrophages, increasing free radical activity, blood clotting mechanisms and impairing fibrinolysis.

References: The new science of growing older without aging BY Dr Philip Lee Miller, life extension foundation edition The Genius ultimate peeling anti-îmbătrânire în două faze solutionby Dr Stephen Sinatra The british journal of pharmacologyMagnesium intake and risk of coronary heart disease among men j Amer coll Nutri ,23 By Al Delaimy et all Zimmer HG, Hibel: the oxidative pentose phosphate pathway in the heart: regulation, physiological significance and implicationsBasic res cardio Multumim oaspetilor nostri, participantilor cat si companiilor prezente la acest eveniment si ai anuntam pe aceasta cale ca in data de mai se va organiza Al Treilea Congres International de Medicina AntiAging si Primul Congres International din Romania de Lasere in Medicina si Chirurgie.

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Cele doua congrese vor avea loc in acelasi timp, sub patronajul Asociatiei de Medicina Anti-Aging, in Bucuresti organizandu-se numeroase workshop-uri atat de medicina anti-aging cat si de estetica si lasere. Toate manifestarile stiintifice vor fi acreditate de Colegiul Medicilor iar participantii vor primi credite EMC.

De asemenea datorita bunelor relatii cu Academia Americana de Medicina Anti-Aging avem bucuria de a va anunta ca initiatorul conceptului de medicina antiaging Dr. Ahmad Najjar The medical use of tattooing is a highly specialized field for the implantation of pigment within the skin in a safe and sterile environment by recognized experts.

Dermografia i procedurile de tatuaj medical Folosirea tatuajului medical este un domeniu de nalt specialitate practicat de ctre exper i care realizeaz implantul pigmen ilor n piele ntr-un mediu steril cu protec ie corespunztoare dermografia. Valoarea tratamentului printr-o metod relativ rapid i simpl de a ob ine rezultate cosmetice satisfctoare de lung durat este dovedit n lotiune demachianta unor pr i anatomice cum ar fi crearea sprncenelor, simularea firelor de pr n alopecie, tricotilomania, refacerea zonelor de pilozitate pierdute n tratamentul de chimioterapie sau datorit tulburrilor genetice.

Camuflarea cicatricilor post operatorii n interven iile chirurgicale pe scalp i craniu, periocular, n excizia leziunilor de carcinom bazocelular demonstreaz eficien a de lung durat a tehnicii de tatuaj medical. Camouflaging post surgical scars in craniosurgery, periocular interventions, excision of basal cell carcinoma lesions demonstrate its long-term effectiveness. Restoration of pigmentary variations of skin: vitiligo, post trauma achromies, burns scars explain why dermography is suggested as an alternative particularly when other medical therapeutic attempts have failed in repigmenting, this often disfiguring conditions.

The initiative contre limmigration suisse anti aging elaborate tattooing technique is used also successfully for correction of facial irregularities after cleft lip and palate surgery.

Dermography is also an easy, quick and effective means to ensure restoration of the nipple-areolar complex. Anatomic and chromatic restoration of nipple-areola complex post mastectomy and mastopexy produces a realistic appearing nipple and significantly improves patient's perception of body image. Medical cosmetic tattoo procedures add more options in esthetic field as therapeutic tools for plastic surgeons in treatment of post operatory scars face lift, tummy tuckstretch marks camouflage for Fitzpatrick skin type IV, V, VI, Tattoolift technique for rejuvenation by blepharopigmentation, eyebrows enhancement, lips contour, full lips in maxim cosmetic results.

The practice of dermography as treatment method in various medical specialties expects professionists to follow certain guidelines with respect to ethics, protocol, genius ultimate peeling anti-îmbătrânire în două faze, equipment and accessories, pigments, necessary material for the rich palette of applications. Refacerea varia iilor pigmentare ale pielii n vitiligo, acromii post traumatice, cicatrici ale arsurilor - explic de ce dermografia este recomandat ca o alternativ n particular cnd alte ncercri terapeutice nu genius ultimate peeling anti-îmbătrânire în două faze reuit.

Tehnica rafinat, elaborat a tatuajului este folosit cu succes pentru corectarea neregularit ilor faciale dup chirurgia buzei de iepure. Dermografia este de asemenea un mijloc uor, rapid i eficient care asigur refacerea complexului areolar i al mamelonului. Refacerea anatomic i cromatic a areolei i a mamelonului dup mastectomie sau mastopexie produce o aparen real a acestei zone mbunt ind semnificativ i percep ia imaginii corpului pacientului.

Procedurile de tatuaj medical cosmetic adaug mai multe op iuni n domeniul esteticii ca instrument terapeutic pentru chirurgii plastici. Tratamentul cicatricilor post operatorii dup liftingul fe ei, abdominoplastie, camuflajul vergeturilor pentru tipul de piele Fitzpatrick IV, V, VI, tehnica Tattoolift de rejuvenare prin blefaropigmentare, intensificare sprncene, contur i mrire de volum al buzelor se finalizeaz cu rezultate cosmetice maxime.

Practicarea dermografiei ca metod de tratament n diverse specialit i medicale implic respectarea unor instruc iuni precise att referitor la etic, ct i la protocol, tehnica, manipularea echipamentului, accesoriilor, pigmen ilor, materialului necesar acestei palete bogate de aplica peeling chimic anti-imbatranire.

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Acesta este testul care detecteaz genomul viral HBV. Bora, Dr. Rusu, Dr. Bora Diabetes mellitus is associated with oxidative and carbonyl stress and accelerated non-enzymatic glycation. These processes impairs endothelial function and may play an important role in the pathogenesis of cardiovascular events. We investigated the in vitro serum lipid oxidation kinetics and the advanced oxidation protein products in elderly patients with type 2 diabetes mellitus for assessing oxidative status and its relationships with other metabolic parameters.

Material and Methods: The kinetics of in vitro copper ions induced lipid oxidation in unfractionated serum was genius ultimate peeling anti-îmbătrânire în două faze by continuous recording of the time-dependence of the oxidation products accumulation at nm during five hours and after 24 hours of the oxidation induction. Advanced oxidation protein products AOPP were determined spectrophotometrically at nm.

Thus, the maximal absorbance of oxidation products accumulation at nm ODmax; 0. The oxidation products accumulation at 24 hours after its inducement with copper ions, as well as the rate of their accumulation at 5 hours have been recorded markant increases in study group, compared with control. The levels of serum advanced oxidation protein products were significant higher in elderly patients with type 2 diabetes mellitus versus control group Conclusions: Our results reveal the incresed oxidative status and the acceleration of oxidative processes in elderly patients with type 2 diabetes, which may contribute to extenssive formation of advanced oxidation protein products, which acts as inflammatory mediators and lead to progression of atherogenic injuries.

Produii de oxidare avansat ai proteinelor i cinetica oxidrii lipidelor la pacienii vrstnici cu diabet de tip 2 Diabetul este asociat cu stresul oxidativ i carbonil, precum i cu intensificarea proceselor de glicare neenzimatic. Aceste procese afecteaz func ia endotelial i pot juca un rol important n patogeneza evenimentelor cardiovasculare. Scopul acestui studiu const n investigarea cineticii de oxidare "in vitro" a lipidelor serice i a acumulrii produilor de oxidare avansat ai proteinelor la pacien i vrstnici cu diabet de tip 2, n vederea determinrii statusului oxidativ i a rela iilor lui cu al i parametri metabolici.

Mască expresă anti-îmbătrânire la domiciliu

Material i metode: Cinetica de oxidare a lipidelor n ser total, nefrac ionat, indus "in vitro" cu ioni de cupru IIa fost urmrit prin nregistrarea continu, n func ie de timp a acumulrii produilor de oxidare la lungimea de und de nm, timp de 5 ore; precum i dup 24 de ore de la inducerea ei. Produii de oxidare avansat ai proteinelor AOPP au fost determina i spectrofotometric la nm. Astfel, absorban a maxim la nm, a acumulrii produilor de oxidare ODmax; 0. Acumularea produilor de oxidare la 24 de ore de la inducerea ei cu ioni de cupru, ca i viteza acumulrii lor dup 5 ore au nregistrat creteri marcante la lotul de studiu, comparativ cu cel de control.

Nivelele serice ale produilor de oxidare avansat ai proteinelor au fost semnificativ crescute la pacien i vrstnici cu diabet de tip 2, fa de grupul de control Concluzii: Rezultatele ob inute relev statusul oxidativ crescut precum i accelerarea proceselor oxidative, la pacien ii vrstnici cu diabet de tip 2, care ar putea contribui la extensiva formare a produilor de oxidare avansat ai proteinelor, care, ac ionnd ca mediatori ai inflama iei, pot conduce la progresia leziunilor aterogene.

Ctlin Enchescu No Needle Mesotehrapy represents a new alternative for micro injection treatment, a solution for anti rejuvenation treatment to the face and body.

The new treatment, called Derma Wave No Needle Mesotherapy, use electrical shocks and has biostimulative effects on tissue. Derma Wave treatment is based on a new technique named Aquaphoresis, and use special electrical waveforms H for reactivity of physiological processes from tissue for washing out the cellulite.

No Needle Mesotherapy is a sure method and represents an alternative and a treatment for face rejuvenation, but also an anti - cellulite treatment. This technique is characterized by a comfortable position of clients during therapy, does not require bandages, time for post operator return, it is pain free and does not require anesthesia.

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After the end of the therapy the accommodation of clients in society is more easily because of a new natural look results to the face and body. No Needle Mesotherapy results are semnificative.

After the end of a complete therapy session, generally after maximum 1 month the clients are pleased and they can observe a high quality of face skin, an increase of face tonus and body tonus. The treatment represents a combination between the photodynamic therapy based on wavelength non invasive laser beam and non invasive mesotherapy.

No Needle Mesotherapy use special technique named Aquaphoresis based on different special current forms which permit the transport of active substances - in mezo layer without use needle. This treatment contributes meaningful to the microcirculation improvement, to the sanguine circulation improvement of treated zones, and on the lymphatic drainage.

Ciomaga Georgeta The high risk for cardiovascular diseases at young ages in autoimmune affections is well known. The mechanism of producing these diseases remains unknown and incites to studies.

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It is demonstrated that in this process much more mechanisms intervene which determine alterations of coagulation, perturbations in the equilibrium and structure of vascular endothelium functions. Ox LDL is promoter of atherosclerosis together with inflammatory and immunological mechanisms which are related to deregulation of lipids and formation of spumous cells.

The atherosclerotic cells contain immunogobulins which recognize ox LDL. Ox LDL stimulates production of auto antibodies of B cells while antibodies dependant of T cell have a protective role. The auto antibodies against ox LDL are present at patients with cardiovascular diseases and have an increased level in carotid atherosclerosis, vascular peripheral diseases and myocardial infarction.

The high prevalence was observed in patients treated with corticosteroids, which indicates also an immune response. The increase of ant-ox LDL antibodies levels is observed at patients with autoimmune diseases in rheumatoid arthritis, diabetes mellitus, uremia, psoriasis, systemic lupus erythematosus, antiphospholipid antibodies syndrome, chronic periaortitis, hepatocellular carcinoma, thalassemia, preeclampsia or eclampsia.

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The native antibodies and anti-idiotype antibodies with IGIV have a stimulating effect of atherosclerosis and proeression of cardiovascular diseases. The administration of IGIV at deficiency of apoE modulates the development of fats and progression of fibro-fats in the atherosclerotic plaque and results the reduction of atherosclerosis. Atherosclerosis, even known from very long time, still remains an enigma. The specific diseases caused by atherosclerosis includes diseases of the coronary artery, the cerebral vascular diseases, thromboembotic strokes, transient ischemic strokes and vascular complications from diabetes mellitus.

The thrombocytes are directly involved in the atherosclerotic process. These contain trigger substances of platelet growth factor and of prostaglandins which stimulate the smooth muscles cells from the wall of the arteries. Fibrinogen, thrombocytes and other coagulation factors aggregate with LDL cholesterol, triglycerides and calcium on the arterial wall, this being the starting point for the development of the atherosclerotic plaque.

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The abnormal aggregation will Bolile Cardiovasculare - risc crescut de deces n boli autoimune Riscul crescut al bolilor cardiovasculare la vrste tinere n bolile autoimune este bine cunoscut. Mecanismul producerii acestora rmne nc necunoscut i incit la studiu.

Este demonstrat c n producerea acestora telefon pentru crema anti-imbatranire avansata vitae mai multe mecanisme care determin perturbri ale coagulrii, perturbri ale echilibrului structurii i func iei endoteliului vascular. Ox LDL este promotor al aterosclerozei mpreun cu mecanismele inflamatorii i imunologice ce in de dereglarea lipidelor i formarea de celule spumoase.

Leziunile aterosclerotice con in imunoglobuline care recunosc oxLDL. Ox LDL stimuleaz produc ia de autoanticorpi ale celulei B n timp ce anticorpii dependen i de celula T au rol protectiv.

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Autoanticorpii mpotriva ox LDL sunt prezen i la pacien ii cu boli cardiovasculare i au nivel crescut n ateroscleroza carotidian, bolile periferice vasculare i infarctul de miocard. Creterea nivelului anticorpilor anti ox-LDL este eviden iat la pacien ii cu boli autoimune n artrita reumatoid, diabet zaharat, uremie, psoriasis, lupus eritematos sistemic, genius ultimate peeling anti-îmbătrânire în două faze anticorpi antifosfolipid, periaortit cronic, carcinomul hepatocelular, beta talasemia i preeclampsia sau eclampsia.

Anticorpii nativi i anticorpii anti-idiotip cu IGIV au efect de stimulare a aterosclerozei i progresie a bolilor cardiovasculare. Administrarea de IGIV la deficien a de apoE moduleaz dezvoltarea grsimilor i progresia fibrogrsimi n placa aterosclerotic i rezult reducerea aterosclerozei. Ateroscleroza, dei cunoscut de foarte mult timp, rmne nc o enigm. Bolile specifice cauzate de ateroscleroz includ bolile arterei coronare, bolile cerebrale vasculare, atacuri trombotice, atacuri tranzitorii ischemice i complica ii vasculare din diabetul zaharat.

Trombocitele sunt implicate direct n procesul aterosclerotic. Acestea con in substan e trigger ale factorului de cretere plachetar i ale prostaglandinelor care stimuleaz celulele muchilor netezi din pere ii arterelor.

Fibrinogenul, trombocitele i al i factori de coagulare agreg cu LDL colesterol, trigliceride i calciu pe peretele arterial, fiind punctul de plecare pentru dezvoltarea plcii aterosclerotice.

La procesul trombotic intervin factorii intrinseci, mai ales genele, factorii de cretere, celulele peretelui vaselor i alte formula cosmetică anti-îmbătrânire elvețiană sanguine.

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Toate acestea interac ioneaz contribuind la procesul trombotic. Agregarea trombocitelor este normal n homeostazie, unde trigger este expunerea plachetelor la matricea subendotelial dup injuria peretelui vaselor.

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At the thrombotic process intrinsic factors, especially genes, growing factors, cells of the vessels arteries and other blood components intervene.

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